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If I understand you correctly, this is what you are calling the Phenotypic Null Hypothesis: that a trait being heritable does not mean it necessarily has a direct genetic cause. Particularly relevant to HBD, my understanding is that you might say that blacks scoring lower on tests might be shown to be heritable, but perhaps that could be because of racism. Since blackness is also genetically heritable, if blackness were to cause them to experience racism which causes their test scores to be lower, then this would be a plausible explanation for why low test scores appear to be genetically heritable in blacks, but it would actually be due to blackness being genetically inherited and that causing low test scores through a more indirect means than low intelligence.
That seems plainly reasonable and true so far as I can tell. I think people are perhaps responding to you defensively because this feels like an isolated demand for rigor or weakmanning directed specifically at HBD, without considering the epistemic failings of hardline blank-slatists which are surely even greater. Also I think that showing a trait to be heritable has to count as weak Bayesian evidence at least in favor of a genetic explanation.
Note that there have been studies trying to control for this, like this admixture study:
Global Ancestry and Cognitive Ability
So black people with 30% European ancestry do better on the Penn Computerized Neurocognitive Battery than those with 10% European ancestry, which is already some remarkably fine-grained racism. But people do talk about "colorism" and so they look at genes linked to skin/hair/eye color, but those genes don't seem to have an impact when ancestry is accounted for.
Note that the anti-HBD crowd (i.e. the Turkheimer side of the debate) have put in some effort to make the author of this publication Bryan J. Pesta, a tenured professor (!), unemployable. I've mentioned this in my first response to tailcalled, but some choice quotes from a Chronicle article praising the cancellation, to understand how the perception of ambiguity in this topic is maintained, indeed how the sausage gets made. It reads very much like a 30's Pravda report on courageous pioneers catching and handling a villain wrecker kulak to the authorities, Scooby Doo style, but that's the clownish reality of the American academia.
[a section on trying to find their vulnerabilities]
etc.
Due to some issues I haven't had the opportunity to engage with the HBD/PNH discussion or indeed to read themotte recently.
That reads like satire, but only a hack would actually name the chief commissar "O’Brien."
And there are people who will look you in the eye and not just defend this behavior, but threaten you for questioning it. Vile.
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Motte: "a trait being heritable does not mean it necessarily has a direct genetic cause"
Bailey: "a trait being heritable is not evidence that it has a direct genetic cause, and the base assumption should be that it does not have a direct genetic cause until very strong evidence exists that it does"
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Yes, that would be one potential example (though I don't expect this to be the true answer because it has been studied by e.g. looking for whether skin color is a mediator, though I'm currently trying to commission a different study which looks at it from a different angle).
However the phenotypic null hypothesis of course applies to tons and tons of behavior genetics, not just within race stuff but also lots of other places. And HBDers often discuss other behavior genetic studies without properly appreciating the phenotypic null hypothesis.
I mean I've definitely called out people on both sides for nonsense. I even tend to hang out in Turkheimer's mentions and critique him. So I don't think I'm doing isolated demands for rigor, I think I'm doing widespread demands for rigor.
The prior probability that a variable is heritable is really really high. Meanwhile, Bayesian updating depends on the probability of the evidence being low, as otherwise it doesn't change your priors much.
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